My notes do not do justice to his presentation. I was busy reading the slides and listening with rapt attention to his fascinating insights rather than taking notes. Therefore, they're really just a few things captured here and there. The Trial Lawyers Association of BC provided a CD with the notes and other information relevant to the conference. Having attended another one of these conferences a few years ago I needed a wagon to carry home the vast collection of notes that different speakers had provided. I thought the CD was considerate given that some of us older sorts had had personal injuries and could well carry home a cd easier than all the binders of previous years.
Myofascial Pain: Relationship between Pain, Impairment and Function
Dr. Mark K. Frob
The Essential Soft Tissue Conference, March 30, 2012
Myofascial pain syndromes are characterized by regional muscular pain patterns typically involving groups of muscles which functionally control complex movements in a specific anatomical area.
14.4% of general population suffer from chronic musculoskeletal pain
myofascial pain syndromes in variaous studies account for 21 % to 93% of pain
History: Myofascial Pain Syndrome
-Dr. Janet Travel and Dr. David Simons 1977
- Dr. Janet Travel served Dr. J. F . Kennedy
- Dr. Janet Travel Clinical Professor of Medicine wrote on trigger points in 80 and 90
-Dr. David Simons - aerospace - work on weightlessness in space
Aerospace medicine
Together two of them - produced what remains the bible of myofascial pain
Myofascial Trigger Points (MTrPs)
taut muscle bands t latnt MTrp to active Mtrp
stress
24 to 54% of asymptomatic individuals have latent trigger points
MTrPs - palpable taut bands, equisitely tender, range of motion of taut muscle limitted
#1 cause - axial skeletal asymmetry
Poor Posture
-fatigue
-sleep deprivation
depression
otherwise radiculopathy, deficiency diseases, hypothyroid,
It presents with a story
Localized muscle tenderness
regionally referred pain
stiffness and limitation of range of motion
sensory distrubance - paresthesia
autonomic phenomena - sweating or decrease, temp changes
recognisable localized knot
‘jump sign’ - patient jumps when you touch
‘twitch response’ - muscle twitches when you touch it
muscle weakness without atrophy
Clinical investigation remarkable by it’s absence
No specific lab tests
infrared or liquid crystal thermography can show increased blood flow at trigger site
Electrical studies may show abn
But we don’t use them in clinical investigation - done as research
Treatment
Needling
drying needling - intramuscular stimulation
trigger point injections - local anesthetics, saline, sterile water, botulinum toxin, corticosteroids
neural acupuncture - injection at the acupuncture points - xylocaine
- if it’s going to work its going to work as first treatment
Massage
Stretching
electrical stimulation
TENS
EMG
etc
Has a rule - if you see benefit but it doesn’t keep getting better after three visits benefit may have peak
Medications
non steroidial anti inflammatory medication and cream
tricyclic antidepressants
Nociceptive VS
Central Pain ()Neuropathic)
20-30% of patients with chronic myofascial pain will have concurnet or comorbid central pain characterics
ie hyperalgeisa
Central Pain Characteristics and comorbidities as described by Dr. Gouw
Pharmacology of Neuropathic Pain
a variety of meds , including cannabinoids
Disability and Impairment
AMA Guides to the Evaluation of Permanent Impairment 5th edition
Activities of Daily Living
self care
communication
physical activitiy
sensory function
non-specialized hand activeity
travel
sexual function
Impairment
= loss of use oor derangement of any body part or organ system or organ function
only those impairments interfering with ADLS
not all impairments interfere with ADL
Disability
=alteratioon of an individual capacity to meet personal social or occupational demands statutory or otherwise
PAIN
-pain is subjective
-pain can exist without tissue damage and tissue damage can exist without pain
a patient can have a well established pain syndrome without identifiable organ deficit
eg migraine
Need to assess credibility and pain behaivour
Need to balance indivdiual self reports and clinical judgement of examiners
Pain Behaviours - non verbal behaviour
- primarily observed
- congruent with established conditions
- consistent over time and situation
- consistent with normal anatomy and physiology
- we’d like to find agreement among caregivers
CHRONIC PAIN DISORDER
-pain exists in more than one anatomic area and warrant clinical attent
-exists past expected treatment
-ccuases clinical distress and or impairment
-psychological factors
-symptons not intentially produced or feigned as in factitious disorder or malingering)
not better accounted by a mood, anxiety or psychotic disorder
Testing Instruments
clearly document history
use of pain related impairment worksheets (PRI’s)
Functional Capacity Evaluation -work simulations
No comments:
Post a Comment