Showing posts with label whiplash. Show all posts
Showing posts with label whiplash. Show all posts

Saturday, March 31, 2012

Chronic Pain and Temporal Mandibular Dysfunction

When people get whiplashes their jaw can be involved. This can lead to chronic pain in face and neck or complicate whiplash pain. Dentists and othodontists have noted this.  Today at the Trial Lawyer's Association of BC Essential Soft Tissue Injury conference a physiotherapist presented on this covering the topic neatly and succinctly with relevant and useful slides.  These are the notes I took and of course they don't cover all the material. The conference offered a CD with the various lecturers notes on that my own notes tend to remind me of what I thought was useful clinically and give a glimpse of what kind of information this conference covered clinically.
Essential Soft Tissue Injury Conference
March 31, 2012
Physical Therapy Treatments for TMD: Giving Hope to your Patients and Clients, Angelica Reeve, MSc,PT, BSc PT MACP
19th Street Physiotherapy Clinica
TMD - Temporalmandibular Dysfunction
TMJ is the jaw joint
connect mandible to temporal bone of skull by articular disc
Opening mouth requires mandible to slide forward and rotate - makes the disc very vulnerable
Muscles of mastication
masseter
temporalis - one of the reasons people complain of headaches
lateral and medial pterygoid
Temporal mandibular dysfunction
-collective term
-muscular
  • intra-articular (inside joint)
  • Combination
5 categories of very minimal to very severe
Causes
trauma -direct trauma, derangement, adhesions
micro trauma -indirect blow, whiplash, bruxism, adhesion
Mechanical dysfunction - trauma, bruxism, muscle imbalance, posture
Osteoarthritis - any of the above
Signs and symptons
loss of range of movement or locking
joint noises
pain - tmj, fascial,earache
pain ful neck
sudden change in bite
decreased hearing, blocked ear
Whiplash - hyperextension and hyperflexion
High incidence of TMD with cervical spine disorders
Five times more likely to develop TMD
20% of recorded cases TMD main complaint
Deep cervical spine muscles can be involved  in whiplash and contribute type i fibers versus type ii more common in superficial muscles
Muscle imbalance
-loss of spatial awareness
posture changes
forward head posture has been found to be correlated to TMD
Treatment
involves positive retraining
deep neck re posturing
Need to taught how to sit upright
Chronic pain
-provides more of a challenge
-change in hard wiring of brain
-increased sensitization
-anxiety and depression
Homunculus in Somatosensory Cortex
Homuncular Man = more face and hand
Chronic TMJ Pain can be associated with problems
  • fascial expression recognition
-two point discrimination
-loss of expression
______________________________________________________

Personal Injury, Chronic Pain and Comorbidities

Dr. Daniel Gouws completed his medical degree at University of Stellenbosch before emigrating to work in Southland, New Zealand, and Saskatchewan before coming to British Columbia. He passed his Master of Sciences at McGill University in Occupational Health Sciences before becoming a board member of the Occupational and Environmental Medical Association of Canada. He is an extremely intelligent, lucid presenter whose clinical experience is obvious from his revealing insights into the care of patients.  His talk, "Comorbidities and Timelines - What to watch when for when your injured patient or client isn't getting better" was a most informative.
The objective was to identify the factors that could contribute to a poor outcome in patients with soft tissue injuries.
The Trial Lawyers Association of British Columbia, Essential Soft Tissue Injury Conference provided a CD with the notes of speakers and other relevant information. I made some notes during the presentation relevant to my own personal interest. It doesn't do true justice to the wealth and breadth of information that Dr. Gouws presented or give you the slides both medical and humorous that accompanied his presentation.  It does give a glimpse and for that reason alone I think it's worth including them here.
Soft Tissue Injury Conference, TSABC, Vancouver Convention Centre
March3012
Comorbidities and Timelines, What to watch for when your injured patient or client isn’t getting better
Dr. Daniel Gouws,
South Africa
Occupational Health McGill
Mechanism of Injury
  • whiplash injuries occur when head accelerates relative to the body, excessive torgue and shear...damage through compression and distraction
Forces involved are considerable, at an imact speed of 20mph (32 km/hr) the human head reachs a peak acceleration of 12 g during extension
If the individual head is in slight rotation, a rear end impact will force the head further into rotation before extension occurs.
In addition to neck pain
-headache
visual disturbances
dizzines
weakness
parasthesiae
cognitive deficits
TMJ
They can often be presenting also with symptons of concussion
Clinical course
Majority of patients improve spontaneously over the first three months
don’t know which go sour
Medical Model
Diagnois
  • History (subjective)
  • Exam (objective)
  • Ass
  • Plan
Differences
Acute pain versus chronic pain
  • chronic pain doesn’t get better
Role of patient with pain is different for acute versus chronic
Acute - follow treatment advice
Chronic pain - ‘partner’ relationship in care , patient responsible for daily management, very different from treatment of appendicitis
Chronic Pain
-lasts more than 3-6 months
varies in intensity from mild to excruciating
one area or multiple areas
emegence of complications
  • increased sensitivity to pain
  • emotional and cognitive distress
  • non restorative sleep
  • fatigue
What is happening in backgrown
  • dogs in background of slides
Chronic Pain
  • chronic co morbidities
Patients get frustrated with us
We get frustrated with them
Blame game
-There is no exact relationship among - degree of pain, extent of pathological change and extent of impairment
-We do not prove or disprove pain with special investigations.  Special investigatiosn are only considered to be appropriate if they will change the management of the patient
-It is inappropriate to request special investigaton in the abscenede of clinical findings such as nerve root entrapment or potentially surgically correctable lesions.
Comorbidities
  • psychosocial
  • what is the effect of the injury and comorbidities
  • deconditioning
  • post traumatic stress
  • substance abuse
  • Depression and anxiety
  • Chronic pain patients who are depressed are 9x more likely to be disabled
  • Untreated psychopathology is greatest likelihood for poor outcome
Loss of Control
  • Regression
  • Isolation
All my friends are dead
Diagnostic Dilemna
  • extensive assessment by multiple physicians
  • numerous investigations (included repeated diagnostic studies) without any real improvement or resolution of symptons
Drugs
  • chronic pain is not an emergency, should get appropriate appointments at appropriate times
  • beware of opiates
  • Canada has distinction of being one of the top prescribers of opiates in the world
Cognitive Distortions
  • unconscious complications of chronic pain
  • non uncommon - we are all prone to cognitive distortions - ex ‘the little engine that couldn’t because he was a worthless bum like your father’
  • emotional reasonling
  • mind reading
  • entitlement
  • ‘la belle indifference’  - patient smiling while saying her pain is 9/10
  • hopelessness
Kinesiophobia
limits activity
limits treatment compliance
becomes self perpetuating - less action - less activity - more pain
Catastrophizing
  • research shows relationship between catastrophising and heightened pain intensity -
Relationship problems
-less interest in sex
Procrastination
-once pain is under control I’ll do more
-I have to wait for my MRI because I can do anything (like take out garbage)
Loss of control
  • fundamental to ability to cope
  • learned helplessness
  • the ability to gain a sesnse of control is fundamental to the ability to cope - self control likened to an energy source and fatigue
  • once you become disabled a lot of that which was taken for granted becomes a bid decision
Workplace Factors
  • adverse outcomes in pain study
  • 1)do you enjoy your job
  • 2) do you get along with your supervisor
Workplace autonomy
  • a factor - disability of chronic pain - less work autonomy - more pain disability
After 6 months chance of return to work drop
Discuss return to work considerations
What can be done?
-individual cognitive behavioural psychotherapy
-exercise
-work conditioning/work hardening
-ergonomic modifications
-modalities used in conjunction with active exercise
  • pain self-management programs
  • PDP, PGAP programs
Elements
-set personal goals
  • improve sleep
  • -increase physical activity
  • manage stress
  • etc
Loss of control
Goal therapy can be effective in helping patients regain control
Chronic Pain Self Management Program
www.selfmanagementbc.ca





Thoracic Outlet Syndrome and Personal Injury

Two truly brilliant doctors presented the most delightful and complex presentations about a not uncommon presentation post trauma and post whiplash.  One was a surgeon. One was a physical medicine specialist.  The slides each had were very carefully chosen and much of their presentations was related to the anatomical drawings and other bits they showed visually.
I was taken back to the very best of the anatomy presentations I had as a medical student coupling that with the latest and greatest of grand rounds presentations. Following that were a question and answer series mostly directed by lawyers.
I knew the anatomy and pathology. I knew the diagnosis. I even knew the general information about treatment though was impressed to get the very latest and even the still controversial aspects of cutting edge management. What I wouldn't have got at medical school or in grand rounds was the lawyers questions. How does this condition apply to a patient with a disability claim. What is the claim and counter claim to the presentation.  Now that's where the doctors answers were most interesting.  It was a bit of a snapshot of a trial really and the doctors did a marvellous job of responding and making their responses understandable to all and sundry. No wonder they are considered the best in their field.

Further I've seen 2 cases this last year myself.  I'm going to review both because I thought one specifically was carpal tunnel syndrome but when everything came back negative I suggested we take a wait and see attitude.  I confess I wish I and the fellows family physician had heard both these presentations because I think there is more I can offer thanks to these fine gentlemen.

These are my notes. The Trial Lawyers Association of British Columbia provided a cd with alot of the papers and presentations. There was so much information being presented that with both doctors I only gleaned highlights.  Further a lot more was on the slides and finally I was only selecting out the bits and pieces of these talks that pertained specifically to my concerns. That said, I think sharing them gives a person a glimpse into the wealth of information these two gentleman presented.  I would strongly recommend anyone with similiar concerns contact them.  In sharing these notes I'm as interested in sharing the level of erudition and discussion that goes on at these conferences.


Post Traumatic Thoracic Outlet Syndrome
Soft Tissue Problem
Dr. A.J. Salvian MD - Surgeon and Vascular Surgeon
 Was a University of Manitoba Vascular Surgeon  (note this man has credentials up the yin yang , they went on for pages and then another speaker took over to continue for two weeks further citing all his accomplishments, I've just extracted out of the long list of international achievements this 2 year stint when he did vascular surgery at University of Manitoba. I did this because these are my notes, I did a month rotation in Vascular Surgery at University of Manitoba. I remember falling asleep holding retractors on livers as the vascular surgeon put in a shunt and did aortic bypass surgery.  At the time the Vascular Surgery department was the leading Canadian service and people were flying in from all over the world surgery .  
Someone else might have noted that he was President of the Canadian Society of Vascular Surgery or that he does surgery at Children's and Vancouver hospital or any number of the accolades that can be referenced to him Yet I noted this fact - it's the way of my note taking. )
Thoracic Outlet Syndrome :  types
  1. Arterial
  2. Venous
  3. Nerurologic
Neurogenic (true)
Post Traumatic 
-Dynamic
-Myogenic
- Disputed
Anterior Scalene Muscle
Brachial Plexus
and Arterial
  • all of them can be compressed
Thoracic Outlet Syndrome 
-Arterial (Major))
Major Arterial (5%)
-Associated with bony abnormality usually
-Aneurysmal dilatation
-distal embolization
-arterial 
thrombosis
  • patients can present with ischemic hand
  • Investigation and see if we can do bypass
Thoracic Outlet Syndrome
  • Venous (5-15%
may or may not be associated with ‘effort thrombosis’
-cyanoused arm, venous distension
-contrast veography
Treatment
-lytic therapy “mist catheter” TPA (urokinase streptokinase) 6 weeks
Thoracic Outlet Syndrome - Neurogenic
History
Dr. Cooper Subclavian Artery thrombosis - 1500
  • cervical rib syndrome
  • 1861 Coote Cervical rib resection
  • 1895 Advent of radiography 
  • 1916 -100 cases of cervical rib syndrome reported
  • early 1900’s focus to other structures
  • 1935 Scalenus anticus syndrome
  • 1943 compression between clavicle and first rib 
  • 1956 ‘thoracic outlet syndrome
A number of operations since then
Present with history of trauma
Headache, neck and midscapular pain
Gradual onset of numbness and tingling, pain from should down the arm, forearm and fingers, - upper plexus C5-6 or lower plexes C8T1.  Often at night and overhead use
Cold blue hand
Thoracic Outlet Syndrome
10% spontaneous
Mostly whiplash injury
Early physio makes them worse
The onset of paraesthesia is usually 1-2 months but can be delayed
Activities that increase neck tension, elevation of arms, lifting or pulling bring on symptons
abscence of use, helps
Often unilateral
GP often thinks it’s carpal tunnel syndrome
Neurologist says nerve conduction studies negative
X rays of C spine often negative or mild
Patients are often told they have nothing or mild CTS
Xrays of c spine are negative or mild . May show some straightening
Patients are developing a “spontaneous nerve’ entrapment.
Predisponsing anomalies are very common
  • 4 large ‘cadaver’ studies - anatomic variance between 35 and 60%
bony or soft tisue
External traum, muscucla spasm, hypotonic shoulder muscles, repetitive injury
Brachial plexus
median C5678 t1
Ulnar c8t1 c7
Radial C5678
Dermatones roots
Trauma and Thoracic Outlet syndrome
37% of ‘whiplash injuries ‘  developed brachial plesuxs irritation dr. idle
35 % have anatomic variations
Thoracic Outlet Syndrome
-neurogenic
cervical rib syndrome
Congenital fibrous bands and ligamentous structures 37 - 63% 
  • 8 types
Histopathology
  • scaring noticed o
Neck Trauma and Thoracic Outlet Syndrome
  1. direct trauma - fracture of rib or clavicule - can have immediate numbness
True thoracic outlet syndrome - no obvious start - pain, paresthesia
Occipital headaches
Thoracic Outlet symptons
-pain shoulder - pain and tenderness, neck and scalene
headache -occipital 74%
-weakness
vascular problems
-irritation of autonomics
true arterial symptons
sympathetic symptons
physical exam
-weak grip strenght
supraclavicular tenderness over scalene 
tinel’s sign
stretch of scalene (hands in air) reproduces paresthesia and pain
arterial obliteration - normal but can indicate anatomic variant - bruit
Differential dx
-radicular-cervical spine nerve root compression -disc , osteophyte
-peripheral nerve compression - carpal tunnel or ulnar entrapment syndrome
-direct brachial plexus injury - stretch, traction,
Diagnosis
Xray of thoracic outlet
Ctscan of thoracic
Nerve conduction to rule out other conditions
Treatment
Conservative therapy
-relax scalene
Stretching, abdominal, breathing, 
-avoid heavy lifting
-botox injection
dominant arm - serious disability
Indication of surgery
-failure of medical management
Post MVA - 80% improvement, 10 % no better, 10% no improvement 10% worse
  • follow up showed benefit took 23 months physical and mental 10 months
Results 
thoracic outlet syndrome - Jamieson Artical, CJS Vol 39, Nov 4, 1996

Over the lunch break a judge had spoken to the important contribution of the physiatrists to the discussion of soft tissue injury and personal injury suits. This tidbit was of course included by the one introducing Dr. Laidlow. 

Soft Tissue Conference
March 30, 2012
Medically Mediating the Disputed Thoracic Outlet Syndrome
Dr. Duncan Laidlow MD FRCPC (physical medicine)
Thoracic Outlet Syndrome
Vascular - venous or arterial
Neurogenic - 98% - neurogenic
-true neurogenic -rare - wasting in arm and -emg show abnormality in this group, in ulnar distribution
  • disputed - we know it exists but can’t prove it
Lack of accepted diagnostic criteria
Lack of measuring standards
Lack of information on history
Research limitations
2010 Cochrane review - only one study worthy of review 1966 study
-compression - in sub scalene space  (two muscles surrounding brachial plexus) these are prone to damage in whiplash injury
T1 fibers are most prone to injury as lowest 
Most common presentation is numbness of inner arm and ring finger
Anomalies of the Thoracic Outlet
  • Only 10% of bilateral ‘normal’ anatomy 
Disputed
lack of agreed clinical features
debate about etiology
lack of confirmatory diagnosis procedure
highly debated treatment outcomes
association with psychiatric issues and secondary gain
Causative
-inherrent predispostion
-trauma
-abnormal posture
Upper and lower varieties
  • arm and hand
  • upper neck and some of face
Diagnostic evaluation
EMG
Alot of physical examination ‘signs
Most commonly employed
Modified upper limb tension test
  • looking for reproduction of signs and symptons
Adson test
-turn head to sign and palm up
Elevated arm stress test
-arms up
Hyperabduction test
-reaching
Eden’s test
-shoulders brought forcibly backwards
Downward pull test 
-pulling arm down.
Plain x ray
CT 
MRI
Anterior Scalene Block
  • helps to decide if surgery will help
Radiculopathy - radiating pain more often, numbness more discrete
Peripheral entrapment neuropathy - just distal fingers
  • if numbness beyond wrist unusual and therefore usually thoracic outlet
Non Operative treatment
  • studies - 65-100 % treatment  (typical 65-80%)
  • but all have all kinds of problems of randomization
Evidence based conservative approach
-avoidance of purely passive measures
-education
-ergonomic adjustments where possible
  • nutritional counselling
  • exercise program - stretching scalene



Wednesday, May 26, 2010

Whiplash Associated Disorders

"Whiplash" is defined as an acceleration-deceleration mechanism of energy transfer to the neck that results from rear-end or or side impact motor vehicle collisions. It can also derive from diving accidents or other similarly occurring impacts that result in bony or soft tissue injuries. These injuries can in turn lead to a variety of clinical manifestations. The collection of clinical manifestations has been called "Whiplash Associated Disorders" or WAD.

The Quebec Task Force WAD classification according to clinical presentation is as follows:

Grade Clinical Presentation

0 No neck complaints and no physical sign(s)

I Neck pain, stiffness or tenderness and no physical sign(s)

II Neck complaint and musculoskeletal sign(s)

III Neck complaint and neurologic sign (s)

IV Neck complaint and fracture or dislocation


The Gargan and Bannister classification of symptom severity is as follows:

Group Symptons

A Asymptomatic

B Mild Symptons not affecting work or leisure activities

C Intrussive symptons interfering with work or leisure. Frequent use of analgesics, orthosis, or physiotherapy.

D Severe problems: lost job, continual reliance on analgesics, orthosis. Repeated medical consultations.

The epidemiology of Whiplash Associated Disorders varies from country to country. The incidence has been reported as highest, 188/100,000 in the Netherlands to lowest, 39/100,000 in Australia.

Early ideal treatment involves the general rules of assessment for major trauma at the initial site and time of the accident. These involve preservation of life and prevention of further damage to the spine and cord and preservation of spinal function. This has been laid out in the US National Acute Spinal Cord Injury Study. The key is to assume spinal instability until proven otherwise.

In the Emergency Department Acute Traumatic Central Cord Syndrome (ATCCS) must be excluded. This is a complex spinal cord syndrome which presents with incomplete neurological deficits such as the ability to walk but not move hands, and sensory changes such as burning hands or urinary retention. MRI generally shows white matter involvement but no hemorrhage. The prognosis is generally good.

Fractures are ruled out by xrays.However if the xrays are normal but the patients persists in having severe pain special x ray views are indicated. If persisting severe pain without any abnormality being found then a hard collar is applied and an MRI is done within24-48 hours showing flexion/extension views to rule out ligamentous injury.These xrays and imaging are all however open to a variety of pitfalls requiring expert interpretation.

Once major injuries are ruled out the normal approach to WAD (Whiplash Associated Disorder) is reassurance and education, no soft collar, Non steroidal anti inflammatory medication such as ibuprofen or naproxens, and early mobilization. Rest and cervical collars can have a detrimental effect on the outcome.

This said nearly 75% of injured patients report immediate symptons such as cervical pain, painful neck movement, painful back, shoulder pain, disturbance of consciousness and dizziness. Examination may show paresthesias, ie unusual sensations, and weakness. Later patients may have visual disturbance, problems with concentration, fatigue, sleep impairment, as well as irritability and anxiety and depression. Interestingly studies show that the anxiety and depression are most directly related to the presence of symptons such as pain and disability. Anxiety and depression do not appear in contrast in those who are early asymptomatic.

While it is important to remember that approximately 10 to 30 % of the general population who have not had an injury report chronic neck symptons, studies show that 15 to 40 % of the WAD patients have chronic neck pain with 10% reporting this as severe. Studies have showed that there can be little alteration of in symptons by three months and stabilizing at 2 years. Authors in prospective study showed little alteration in sympton severity for the majority of patients (64%) between 3 months and 7.5 years . Between 3 months and 2 years the symptons fluctuated significantly and prognosis based on this were unreliable. Therapeutically the greatest benefit for influencing outcome was in those first three months. One interesting study showed that high dose methylprednisone resulted in earliest return to work.

There have been many issues raised relative to the prognosis or eventual outcome of the injury. This has been difficult because there is no clear definition of what is recovery. At best crude measures of symptons or disability have been used along with such matters as 'return to work', 'discontinuation of treatment', or 'conclusion of litigation' .

That said, a systematic review of prospective cohort studies done in Pain 2003 concluded that "strong evidence was found for high initial pain intensity, and strong evidence for no prognostic value for older age, female gender, high acute psychological response, angular deformity of the neck, rear-end collision and compensation". Limitted evidence was found for some physical, psychosocial, neuropsychological, crash related and treatment related factors in terms of prognosis.

References:

Initial assessment of whiplash patients, Dr. R Gunzburg, M. Szpalski, J. Van Goethem,Pain Res Manage Vol 8 No 1 Spring 2003

Fluctuation in recovery following whiplash injury, 7.5 year prospective review, P.J. Tomlinson, M.F. Gargan, G.C. Bannister, Injury, Int. J. Care Injured (2005) 36, 758-761

Sensory hypersensitivity occurs soon after whiplash injury and is associated with poor recovery, Michele Sterling, Gwendolen Jull, Bill Vicenzino, Justin Kenardy, Pain 104 (2003) 5009-517

Prognostic factors of whiplash-associated disorders: a systematic review of prospective cohort studies, Gwendolijne G.M. Scholten-Peeters, Arianne P. Verhagen, Geertruida E. Bekkering, Danielle A.W.M. van der Windt, Les Barnsley, Rob A.B. Oostendorp, Erik J.M. Hendriks, Pain (104 (2003)303-322